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NAA exerts its function in a histone acetyltransferase P300/CBP-associated factor (PCAF)-dependent manner, leading to lamin A acetylation, impairing its interaction with Sun2 (Sad1 and Unc84 domain-containing protein 2) and subsequent lytic granules, from CD8 + T cells and NK cells, polarization and release [214]

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This may be a beneficial effect (bypassing key), resolving a wide range of occlusion/occlusion-like syndromes vessels and multiple organ failures (for review, see [24]) by re-establishing blood flow through the activation of the collateral additional vascular pathways

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